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A little bit about steroids: aromatizing and not only.

Prolonged use of steroids can lead to increased levels of estradiol in the blood and as a consequence of it - to aromatization. I note here that this property have only the so-called aromatizable steroids. Testosterone is considered to have the biggest incline to this process, of course. The second place occupies such a steroid as methandrostenolone. Boldenone and flyuksimetreon have more weak property of aromatization.

There are steroids that can be converted to estradiol without the process of aromatization, these include nandrolone, norethandrolone and ethylestrenol. However, it makes no significant relief, and the level of estrogen from these drugs is increased in any case.

It should also be noted that among the non-aromatizing drugs there are the so to say outcasts. I mean the drugs that can cause excessive accumulation of fluid in the area of the men nipples without estrogens. This is primarily oksimethalon.

But let's go back a bit and talk about the following. Is it possible that the level of estrodiol in the blood of a men will rise even with a short-term use of steroid drugs? No it is not possible. As with any side effect, this defect requires prolonged use or overdose.

And the last thing I would like to discuss - each person has an individual reaction to the estrogens in the blood because we are all different as banal as it may sound, some people even have immunity to the process of aromatization, and some on the contrary may be particularly inclined to this problem.



Restoring the natural balance.

Lets finish with the theory and go back to what we actually started with and what I wanted to tell you about, that is to anti-estrogens. It is a mistake to assume that anti-estrogen is a drug created specifically for men, as if it was created to suppress the development of the feminine in men. The situation is quite opposite, originally anti-estrogens were created for women who had problems with an excess of estrogen in their bodies. For example, particularly large amounts of estrogenic hormones can lead to infertility in women. However, such cases are very rare, and not more than a hundred of such cases are registered per year.

Now let's talk about anti-estrogens themselves. They are divided according to their characteristics and properties into two groups. The first group are the aromatose inhibitors. The second group are the estrogen receptors blockers. In the first case, aromatose inhibitors are joined to the aromatose enzymes, sorry for the tautology, just thr same as testosterone does and thus reduce the amount of these enzymes to minimum and reduce the risk of developing of aromatization.

Blockers act quite different, they join to estrogen receptors without activating them, but quite contrary, they begin to act, creating a disturbance and interfering estrodiol to perform its functions.

That is, we see that in the first case anti-estrogens simply stop conversion of testosterone to estradiol, and in the second case estradiol simply stops effect or is blocked.

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